A new article in Nature has identified the molecular triggers that have given rise to the global epidemic of autism.
The article is the first of its kind to describe a novel molecular trigger that is also known as a “master-in-biology” (MIM) gene, which is a group of genes that control the development of specific proteins.
These proteins control the growth and function of the brain, heart, pancreas and other organs.
The study’s lead author is Professor Yannis Georgopoulos from the Department of Molecular and Cellular Biology at University College London, who described the MIM gene as the key to autism’s genetic origins.
“The MIM genes are a group that control growth and functions of the nervous system,” Professor Georgopoulos told Al Jazeera.
“In other words, they are important for the development and maintenance of a nervous system that is important for survival.”
The MIMs have a large number of functions in different brain regions.
Professor Georgopolous said that the Mim genes are involved in a number of other aspects of the disease, including autism spectrum disorders, epilepsy, Alzheimer’s disease and other conditions.
Autism spectrum disorders and epilepsy are two of the leading causes of disability worldwide.
Professor Georgeopoulos said that autism is a complex disorder with a multitude of genetic and environmental causes.
He said that it is also linked to the development in the brain of a number different genes.
“One of the genes that is involved in autism is called AUTN1,” he said.
“That is the gene that has been shown to have a role in the development, development and transmission of autism spectrum disorder.”
Professor Georgoulopoulos explained that the autism spectrum is defined by symptoms such as hyperactivity, poor social skills and repetitive behaviours.
“We know that there are many other genes that are involved,” he told Al Jersualt.
“Some of these are involved with motor and sensory function, some of them are involved only in the language and visual functions.”
He said the researchers wanted to identify which of these genes were responsible for the spread of autism across the world.
The Mim gene, Professor Georgosopoulos said, was not the only one involved in the spread and spread of the autism.
“It is the only MIM that we know of that has not been found in a previous population,” he explained.
“So we think it is a really important one.”
The researchers studied the genomes of more than 3,000 people and compared their MIM variants with other known autism genes, to see if they shared a common mutation that caused autism.
They found that the mutation, called H4K1T2, causes the Mims to be more closely linked to other autism genes.
This suggests that the mutations in the Mimbys were a result of the mutation.
However, there are other mutations that have not been identified that could be involved in some of the disorders in the autism gene pool.
The team also discovered that the different variants in the human Mimby were more similar to each other than they were to other genes.
That suggests that one mutation in the mutation was responsible for some of these disorders.
Professor Alastair MacFadyen, a leading autism researcher at the University of Sheffield, said the findings were exciting and important.
“These results reveal that the spread in autism has a strong genetic basis, with the mutation H4k1T1 being the most likely candidate,” Professor MacFadieen told Aljazeera.
“H4k is associated with a number other autism-related disorders and it has also been found to be involved with the genetic basis of schizophrenia.”
Professor Macfadyen told The Sunday Times that it was exciting to find that the two mutations that are responsible for autism could be associated with the same genetic basis.
“There are many genes that influence autism and the Mimm genes are just one of those,” he added.
“This finding is exciting, as the Mimer genes were not identified until now.”
The research is published in the journal Science.